Acute increases in intramuscular inflammatory cytokines are necessary for the development of mechanical hypersensitivity in a mouse model of musculoskeletal sensitization
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Animal Diseases - Disease Models, Animal
Animal Population Groups - Mice, Knockout
Animals
Antigens, Surface - Receptors, Tumor Necrosis Factor, Type I
Biological Factors - Cytokines
Biological Factors - Interleukin-1
Biological Factors - Interleukin-1
Biological Factors - Interleukin-6
Biomarkers - Receptors, Tumor Necrosis Factor, Type I
Male
Mice
Mice - Mice, Knockout
Mice, Mutant Strains - Mice, Knockout
Models, Animal - Disease Models, Animal
Models, Biological - Disease Models, Animal
Muscle, Striated - Muscle, Skeletal
Musculoskeletal Diseases - Myalgia
Musculoskeletal Diseases - Myositis
Musculoskeletal System - Muscle, Skeletal
Nervous System Diseases - Hyperalgesia
Nervous System Diseases - Myalgia
Nervous System Physiological Phenomena - Myalgia
Neuromuscular Diseases - Myalgia
Neuromuscular Diseases - Myositis
Peptides - Cytokines
Peptides - Interleukin-1
Peptides - Interleukin-1
Peptides - Interleukin-6
Proteins - Cytokines
Proteins - Interleukin-1
Proteins - Interleukin-1
Proteins - Interleukin-6
Psychological Phenomena and Processes - Myalgia
Receptors, Death Domain - Receptors, Tumor Necrosis Factor, Type I
Receptors, Interleukin-1
Receptors, Tumor Necrosis Factor - Receptors, Tumor Necrosis Factor, Type I
Signs and Symptoms - Hyperalgesia
Signs and Symptoms - Myalgia
Date in CU Experts
September 25, 2017 12:41 PM
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