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- Activation of valvular interstitial cells is mediated by transforming growth factor-beta 1 interactions with matrix molecules Journal Article
- Cardiac myosin heavy chains lacking the light chain binding domain cause hypertrophic cardiomyopathy in mice. Journal Article
- Cardiac valve cells and their microenvironment-insights from in vitro studies Journal Article
- Characterization of Cell Subpopulations Expressing Progenitor Cell Markers in Porcine Cardiac Valves Journal Article
- Characterization of valvular interstitial cell function in three dimensional matrix metalloproteinase degradable PEG hydrogels Journal Article
- Designing scaffolds for valvular interstitial cells: Cell adhesion and function on naturally derived materials Journal Article
- Dynamic stiffening of poly(ethylene glycol)-based hydrogels to direct valvular interstitial cell phenotype in a three-dimensional environment Journal Article
- Hydrogels preserve native phenotypes of valvular fibroblasts through an elasticity-regulated PI3K/AKT pathway Journal Article
- Microarray analyses to quantify advantages of 2D and 3D hydrogel culture systems in maintaining the native valvular interstitial cell phenotype Journal Article
- Myofibroblastic activation of valvular interstitial cells is modulated by spatial variations in matrix elasticity and its organization Journal Article
- Quantifying heart valve interstitial cell contractile state using highly tunable poly(ethylene glycol) hydrogels Journal Article
- Role of cell-matrix interactions on VIC phenotype and tissue deposition in 3D PEG hydrogels Journal Article
- Roles of transforming growth factor-beta 1 and OB-cadherin in porcine cardiac valve myofibroblast differentiation Journal Article
- Serum deprivation improves seeding and repopulation of acellular matrices with valvular interstitial cells Journal Article
- Task force on strategic research direction - Basic science subgroup - Key science topics report Journal Article
- Valvular myofibroblast activation by transforming growth factor-β -: Implications for pathological extracellular matrix remodeling in heart valve disease Journal Article