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- G3BP1-dependent condensation of translationally inactive viral RNAs antagonizes infection. Journal Article
- Identification of small molecule inhibitors of G3BP-driven stress granule formation. Journal Article
- Norovirus infection results in eIF2α independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation Journal Article
- Novel stress granule-like structures are induced via a paracrine mechanism during viral infection Journal Article
- RNA partitioning into stress granules is based on the summation of multiple interactions Journal Article
- UBAP2L Forms Distinct Cores that Act in Nucleating Stress Granules Upstream of G3BP1 Journal Article