Glial proinflammatory cytokines mediate exaggerated pain states: Implications for clinical pain
Journal Article
Overview
publication date
- January 1, 2003
has subject area
- Animals
- Biological Factors - Cytokines
- Biological Factors - Interleukins
- Biological Factors - Tumor Necrosis Factor-alpha
- Biological Factors - Tumor Necrosis Factor-alpha
- Blood Proteins - Tumor Necrosis Factor-alpha
- Brain
- Cells - Astrocytes
- Cells - Microglia
- Cells - Neuroglia
- Humans
- Inflammation
- Models, Neurological
- Nervous System - Astrocytes
- Nervous System - Microglia
- Nervous System - Neuroglia
- Nervous System Diseases - Hyperalgesia
- Nervous System Diseases - Peripheral Nerve Injuries
- Peptides - Cytokines
- Peptides - Interleukins
- Peptides - Tumor Necrosis Factor-alpha
- Peptides - Tumor Necrosis Factor-alpha
- Peripheral Nerves
- Peripheral Nervous System Diseases - Peripheral Nerve Injuries
- Proteins - Cytokines
- Proteins - Interleukins
- Proteins - Tumor Necrosis Factor-alpha
- Proteins - Tumor Necrosis Factor-alpha
- Proteins - Tumor Necrosis Factor-alpha
- Signs and Symptoms - Hyperalgesia
- Spinal Cord
- Wounds and Injuries - Peripheral Nerve Injuries
Full Author List
- Watkins LR; Milligan ED; Maier SF
published in
Additional Document Info
start page
- 1
end page
- 21
volume
- 521