Morphological and functional alterations in ventricular myocytes from male transgenic mice with hypertrophic cardiomyopathy
Journal Article
Overview
publication date
has subject area
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Adenosine Triphosphatases - Myosin Heavy Chains
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Adenosine Triphosphatases - Ventricular Myosins
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Animal Population Groups - Mice, Transgenic
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Animals
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Biological Factors - Calcium
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Cardiomyopathies - Cardiomyopathy, Hypertrophic, Familial
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Cardiovascular Physiological Phenomena - Diastole
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Cardiovascular Physiological Phenomena - Myocardial Contraction
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Cardiovascular Physiological Phenomena - Systole
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Cytoskeletal Proteins - Myosin Heavy Chains
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Cytoskeletal Proteins - Ventricular Myosins
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Diacetyl
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Diagnostic Imaging - Microscopy, Electron
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Elements - Calcium
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Environment and Public Health - Age Factors
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Gene Expression
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Genetic Diseases, Inborn - Cardiomyopathy, Hypertrophic, Familial
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Health Care Quality, Access, and Evaluation - Age Factors
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Heart - Myocytes, Cardiac
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Heart Valve Diseases - Cardiomyopathy, Hypertrophic, Familial
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Heart Ventricles
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Investigative Techniques - Microscopy, Electron
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Macromolecular Substances - Myosin Heavy Chains
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Macromolecular Substances - Ventricular Myosins
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Male
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Metals - Calcium
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Mice
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Mice - Mice, Transgenic
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Models, Animal
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Muscle Cells - Myocytes, Cardiac
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Muscle Cells - Sarcomeres
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Muscle Cells - Sarcomeres
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Muscle Contraction - Diastole
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Muscle Contraction - Myocardial Contraction
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Muscle Contraction - Systole
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Muscle Proteins - Myosin Heavy Chains
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Muscle Proteins - Ventricular Myosins
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Muscle Relaxation - Diastole
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Muscle, Striated - Myocytes, Cardiac
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Muscle, Striated - Sarcomeres
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Mutagenesis - Amino Acid Substitution
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Mutation, Missense
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Organelles - Sarcomeres
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Phenotype
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Protein Engineering - Amino Acid Substitution
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Protein Isoforms
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Ventricular Dysfunction, Left
Full Author List
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Olsson MC; Palmer BM; Stauffer BL; Leinwand LA; Moore RL
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