Decreased insulin-receptor signaling promotes the autophagic degradation of β-amyloid peptide in C-elegans
Journal Article
Overview
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Amyloidogenic Proteins - Amyloid beta-Peptides
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Animal Diseases - Disease Models, Animal
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Animal Population Groups - Animals, Genetically Modified
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Animals
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Autophagy
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Biochemical Phenomena - Signal Transduction
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Brain Diseases - Alzheimer Disease
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Caenorhabditis elegans
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Cell Physiological Phenomena - Signal Transduction
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Diagnostic Imaging - Microscopy, Fluorescence
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Green Fluorescent Proteins
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Humans
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Investigative Techniques - Microscopy, Fluorescence
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Lysosomes
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Membrane Proteins - Amyloid beta-Peptides
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Models, Animal - Disease Models, Animal
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Models, Biological
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Models, Biological - Disease Models, Animal
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Muscle, Striated - Muscle, Skeletal
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Musculoskeletal System - Muscle, Skeletal
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Neurocognitive Disorders - Alzheimer Disease
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Organisms, Genetically Modified - Animals, Genetically Modified
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Peptide Fragments
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Peptides - Amyloid beta-Peptides
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Protein-Tyrosine Kinases - Receptor, Insulin
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RNA Interference
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Receptors, Cell Surface - Receptor, Insulin
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Receptors, Pancreatic Hormone - Receptor, Insulin
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Tauopathies - Alzheimer Disease
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Date in CU Experts
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September 9, 2013 9:28 AM
Full Author List
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Florez-McClure ML; Hohsfield LA; Fonte G; Bealor MT; Link CD
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