RCAN1 overexpression promotes age-dependent mitochondrial dysregulation related to neurodegeneration in Alzheimer's disease
Journal Article
Overview
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Abnormalities, Multiple - Down Syndrome
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Aging
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Animal Diseases - Disease Models, Animal
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Animal Population Groups - Mice, Transgenic
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Animals
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Behavior and Behavior Mechanisms - Memory Disorders
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Brain
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Brain Diseases - Alzheimer Disease
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Congenital Abnormalities - Down Syndrome
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Cytoskeletal Proteins - Dynamins
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Cytoskeletal Proteins - tau Proteins
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DNA-Binding Proteins
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Environment and Public Health - Cohort Studies
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GTP Phosphohydrolases - Dynamins
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Genetic Diseases, Inborn - Down Syndrome
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Health Care Evaluation Mechanisms - Cohort Studies
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Humans
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Investigative Techniques - Cohort Studies
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Long-Term Potentiation
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Male
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Metabolism - Oxidative Stress
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Mice - Mice, Transgenic
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Models, Animal - Disease Models, Animal
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Models, Biological - Disease Models, Animal
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Muscle Proteins
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Nerve Degeneration
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Nerve Tissue Proteins - tau Proteins
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Nervous System Diseases - Down Syndrome
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Nervous System Diseases - Memory Disorders
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Neurocognitive Disorders - Alzheimer Disease
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Organelles - Mitochondria
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Peptides - Intracellular Signaling Peptides and Proteins
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Proteins - Intracellular Signaling Peptides and Proteins
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Signs and Symptoms - Memory Disorders
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Stress, Physiological - Oxidative Stress
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Subcellular Fractions - Mitochondria
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Tauopathies - Alzheimer Disease
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Vesicular Transport Proteins - Dynamins
Date in CU Experts
Full Author List
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Wong H; Levenga J; Cain P; Rothermel B; Klann E; Hoeffer C
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