Cyclin D2 is a critical mediator of exercise-induced cardiac hypertrophy
Journal Article
Overview
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Adaptation, Biological - Adaptation, Physiological
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Animal Population Groups - Mice, Transgenic
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Animals
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Behavior - Motor Activity
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Biochemical Phenomena - MAP Kinase Signaling System
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Biochemical Phenomena - Phosphorylation
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Cell Cycle Proteins - Cyclin D2
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Cell Physiological Phenomena - MAP Kinase Signaling System
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Heart - Myocytes, Cardiac
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Heart Diseases - Cardiomegaly
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Hypertrophy - Cardiomegaly
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Metabolic Networks and Pathways - MAP Kinase Signaling System
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Metabolism - Phosphorylation
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Mice - Mice, Transgenic
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Models, Animal
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Multiprotein Complexes - Glycogen Synthase Kinase 3
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Muscle Cells - Myocytes, Cardiac
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Muscle, Striated - Myocytes, Cardiac
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Musculoskeletal Physiological Phenomena - Motor Activity
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Organic Chemistry Phenomena - Phosphorylation
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Peptides - Cyclin D2
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Peptides - Glycogen Synthase Kinase 3
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Physiological Phenomena - Adaptation, Physiological
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Proline-Directed Protein Kinases - Glycogen Synthase Kinase 3
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Protein-Serine-Threonine Kinases - Glycogen Synthase Kinase 3
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Proteins - Cyclin D2
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Proteins - Glycogen Synthase Kinase 3
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Proteins - Glycogen Synthase Kinase 3
has restriction
Date in CU Experts
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September 6, 2013 4:09 AM
Full Author List
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Luckey SW; Haines CD; Konhilas JP; Luczak ED; Messmer-Kratzsch A; Leinwand LA
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