Rectification of muscle and nerve deficits in paralyzed ryanodine receptor type 1 mutant embryos
Journal Article
Overview
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Animal Population Groups - Mice, Knockout
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Animals
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Animals, Inbred Strains - Mice, Inbred C57BL
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Biochemical Phenomena - Synaptic Transmission
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Biological Factors - Calcium
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Carrier Proteins - Ryanodine Receptor Calcium Release Channel
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Cell Physiological Phenomena - Synaptic Transmission
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Cells - Motor Neurons
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Electrophysiological Phenomena - Synaptic Transmission
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Elements - Calcium
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Elements - Potassium
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Elements - Potassium
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Intercellular Junctions - Neuromuscular Junction
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Membrane Proteins - Ryanodine Receptor Calcium Release Channel
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Membrane Proteins - Ryanodine Receptor Calcium Release Channel
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Metals - Calcium
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Metals - Potassium
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Metals - Potassium
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Mice
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Mice - Mice, Inbred C57BL
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Mice - Mice, Knockout
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Mice, Mutant Strains - Mice, Knockout
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Muscle Contraction
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Muscle Proteins - Ryanodine Receptor Calcium Release Channel
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Muscle, Striated - Muscle, Skeletal
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Musculoskeletal Diseases - Muscle Weakness
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Musculoskeletal Physiological Phenomena - Locomotion
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Musculoskeletal System - Muscle, Skeletal
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Nerve Endings - Neuromuscular Junction
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Nerve Tissue
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Nervous System - Motor Neurons
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Nervous System - Neuromuscular Junction
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Nervous System Diseases - Muscle Weakness
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Nervous System Physiological Phenomena - Synaptic Transmission
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Pathologic Processes - Muscle Weakness
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Physiological Phenomena - Locomotion
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Receptors, Cholinergic
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Respiration
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Signs and Symptoms - Muscle Weakness
has restriction
Date in CU Experts
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February 13, 2018 11:03 AM
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