TNF-alpha blockade by a dimeric TNF type I receptor molecule selectively inhibits adaptive immune responses
Journal Article
Overview
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Animals
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Animals, Inbred Strains - Mice, Inbred BALB C
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Animals, Inbred Strains - Mice, Inbred C57BL
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Animals, Inbred Strains - Mice, Inbred DBA
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Antigens, Surface - Antigens, CD
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Biological Factors - Tumor Necrosis Factor-alpha
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Biological Factors - Tumor Necrosis Factor-alpha
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Biomarkers - Antigens, CD
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Blood Proteins - Immunoglobulin G
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Blood Proteins - Tumor Necrosis Factor-alpha
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Chemical Phenomena - Dimerization
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Escherichia coli Infections
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Female
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Globulins - Immunoglobulin G
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Humans
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Immunity
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Immunity, Cellular
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Immunoproteins - Immunoglobulin G
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Listeriosis
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Metabolism - Dimerization
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Mice
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Mice - Mice, Inbred BALB C
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Mice - Mice, Inbred C57BL
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Mice - Mice, Inbred DBA
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Peptides - Tumor Necrosis Factor-alpha
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Peptides - Tumor Necrosis Factor-alpha
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Proteins - Tumor Necrosis Factor-alpha
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Proteins - Tumor Necrosis Factor-alpha
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Proteins - Tumor Necrosis Factor-alpha
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Rats
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Rats, Sprague-Dawley
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Receptors, Death Domain - Receptors, Tumor Necrosis Factor, Type I
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Receptors, Tumor Necrosis Factor
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Receptors, Tumor Necrosis Factor - Receptors, Tumor Necrosis Factor, Type I
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Recombinant Proteins
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Staphylococcal Infections
has restriction
Date in CU Experts
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September 3, 2013 11:11 AM
Full Author List
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Colagiovanni DB; Suniga MA; Frazier JL; Edwards CK; Fleshner M; McCay JA; White KL; Shopp GM
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